APPLY FOR FUNDING
Al and Val Rosenstrauss Fellowship
(5‐10 years post‐doc)Open 1 April 2024
Close 5pm, 30 April 2024 (AEST)
APPLICATION GUIDELINES SCIENTIFIC ADVISORY COMMITTEERebecca Cooper Fellowship
(10‐15 years post‐doc)Open 1 August 2024
Close 5pm, 30 August 2024 (AEST)
APPLICATION GUIDELINES SCIENTIFIC ADVISORY COMMITTEEMEET OUR FELLOWS
FEATURED RESEARCH
Neuropeptide-based approach to improve myelin repair in Multiple Sclerosis
A/Prof Alessandro Castorina
Multiple Sclerosis (MS) occurs when myelin, the insulating layer which forms around nerves, allowing electrical signals to transmit along nerve cells in the CNS, is attacked and damaged by our own immune system. Finding a method to repair myelin damage and proactively protect myelin producing cells from primary damage (neuroprotection) is therefore essential to slowing, treating and eventually curing MS.
View MoreChanging the Landscape of Thyroid Cancer Management
Dr Matti Gild
The incidence of thyroid cancer has increased in the past few decades. There are now more patients who need nuanced risk stratification of their disease. Whilst most cases are associated with excellent prognosis, in aggressive differentiated thyroid cancer (DTC), 25% of cases will develop locally recurrent disease requiring additional treatments. This project's goal was to combine the molecular data with clinicopathological risk factors in order to develop a comprehensive algorithm for personalised treatment in thyroid cancer and make this accessible on a national scale. This could revolutionise thyroid cancer management and other malignancies as the concept is adaptable to other cancer types.
View More“Is a brain peptide sympathetically driving cardio-metabolic dysfunction following intermittent hypoxia?”
Dr Melissa Farnham
Obstructive sleep apnoea (OSA) is highly prevalent and characterised by intermittent episodes of hypoxia during sleep. People with OSA are extremely likely to also have hypertension and/or diabetes. The two overarching issues are: 1. What role does the brain play in the pathogenesis of disease in OSA patients? and, 2. Is there a causal relationship between OSA and the development of hypertension and diabetes?
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